EC Neurology

Brain contusions can be dynamic and expansive, leading to delayed neurological deterioration, with cerebral edema being the main cause of death in this case.

Our work aims to study the epidemiological, clinical, paraclinical, therapeutic and evolving data on post-traumatic brain contusions in order to define the main problems posed by these head trauma patients.

The physiopathology of TBI describes two mechanisms: TBI with direct impact on the skull or TBI without direct impact where the head is subjected to a deceleration/acceleration effect.

We distinguish between primary lesions, immediately present after the trauma and which are inevitable, and secondary lesions which develop in a delayed manner over a few hours and which must be detected and treated.

Brain contusion is an imprecise term encompassing all brain lesions linked to the transmission of the shock wave. They become complicated by ICH and progress towards the onset or worsening of a coma, which justifies their treatment in a center specialized in neurotraumatology regardless of the initial clinical state.

Cerebral edema complicates most parenchymal lesions; it can be localized or diffuse.

Traumatic brain contusions are a major cause of mortality in developed countries. It is the most important structural lesion of the brain, it is complex and dynamic and results from a primary lesion.

 Keywords: Brain Contusion; Cerebral Edema; Head Trauma; TBI

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